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The systemic inhibition of the terminal complement system reduces neuroinflammation but does not improve motor function in mouse models of CMT1A with overexpressed PMP22. Iliana Michailidou, Jeroen Vreijling, Matthijs Rumpf, Maarten Loos, Bastijn Koopmans, Nina Vlek, Nina Straat, Cedrick Agaser, Thomas B. Kuipers, Hailiang Mei, Frank Baas, Kees Fluiter. doi.org/10.1016/j.crneur.2023.100077 Curr Res Neurobiol. 2023

Epileptiform activity and cognitive deficits in SNAP-25(+/-) mice are normalized by antiepileptic drugs. Corradini, Loos et al., Cereb Cortex. 2014; 24:364, PMID: 23064108

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Functional brain defects in a mouse model of a chromosomal t(1;11) translocation that disrupts DISC1 and confers increased risk of psychiatric illness. Bonneau M, Sullivan STO, Gonzalez-Lozano MA, Baxter P, Gautier P, Marchisella E, Hardingham NR, Chesters RA, Torrance H, Howard DM, Jansen MA, McMillan M, Singh Y, Didier M, Koopmans F, Semple CA, McIntosh…

Assessment of Behavioral Characteristics With Procedures of Minimal Human Interference in the mdx Mouse Model for Duchenne Muscular Dystrophy. Engelbeen S, Aartsma-Rus A, Koopmans B, Loos M, van Putten M. Front Behav Neurosci. 2021 Jan 20;14:629043. doi: 10.3389/fnbeh.2020.629043. PMID: 33551769; PMCID: PMC7855581. https://pubmed.ncbi.nlm.nih.gov/33551769/ Keywords: Duchenne, neuromuscular disorder, muscle disease